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Among children with NCI standard-risk T-cell ALL, the EFS for those treated on and studies was inferior to the EFS for those treated on the POG-9404 study.

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From the collective analysis of these studies, imatinib mesylate appears to effectively induce high CHR and cytogenetic response rates with relatively few side effects.

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Physicians can diagnose surreptitious thyroid hormone intake by a low thyroglobulin and normal radioactive iodine uptake scan; subclinical hyperthyroidism can be diagnosed by a low TSH and elevated T3 or free T4 levels.

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HSCT compared with chemotherapy alone was observed in patients with T-cell phenotype (any age) and B-precursor patients younger than 6 years.

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The following adverse reactions have been reported during clinical trials of imatinib mesylate.

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During the first cycle of therapy or at times of worsening disease before intrapatient dose escalation, patients were allowed to receive cytoreductive therapy (leukaphereses and hydroxyurea) to control elevated counts of blasts, platelets, or both.

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CML): in newly-diagnosed adults and children in chronic phase; in patients in blast crisis, accelerated phase, or chronic phase after failure of interferon-alpha therapy.

The data as of January 31, 2002, were then submitted to the health authorities of countries participating in the study as part of the approval process for the use of imatinib in newly diagnosed chronic-phase CML.

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In general, treatment of these diseases is tailored to the manifestations, myeloproliferative or myelodysplastic, that predominate in the individual patient.

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Other than this there are four major mainstays of treatment in CML: treatment with tyrosine kinase inhibitors, myelosuppressive or therapy (to counteract the during early treatment), and treatment.

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Imatinib is a 2-phenylaminopyrimidin derivate () and was initially developed as a specific platelet-derived growth factor receptor (PDGFR) inhibitor, but was later found to inhibit autophosphorylation of Abl and c-Kit.

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The longer-term goals are to decrease or get rid of the cells with the Philadelphia chromosome to slow down or prevent the disease from moving to blast crisis.

The advent of imatinib already appears to have had an impact on the numbers of allografts being performed, and the choice between drug therapy and transplantation for newly diagnosed CML is becoming increasingly difficult.

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